Describing rashes

A rash is a change in the colour or texture of the skin and as such reflects the nature and pattern of a collection of individual lesions. A major consideration in dermatological assessment is to determine the specific patterns of such rashes and their specific characteristics as an aid to diagnosis. Some of the dimensions of skin assessment are specified in Table 3.3. These include site, colour, acuity, surface features and type of lesion.
   
 
Table 3.3 A framework for skin assessment and lesion description.

 1. Site

2. Erythematous, i.e. reddened skin (blanches on pressure) or non-erythematous

3. Acute (<2 weeks duration) or chronic (>2 weeks duration)

4. Surface features
    a. Normal/smooth (i.e. same as surrounding skin)
    b. Scaly
    c. Hyperkeratotic
    d. Warty
    e. Crust
    f. Exudate
    g. Excoriated
5. Type of lesion
    a. Flat: macules and patches
    b. Raised: papules, plaques and nodules
    c. Fluid-filled: vesicles, bullae and pustules
    d. Surface broken: erosions, ulcers and fissures
If non-erythematous describe the

6. Colour
    a. Due to blood: pink, purple, mauve
    b. Due to pigment: brown, black and blue
    c. Due to lack of blood/pigment: white
    d. Other colours: yellow, orange, grey
 
 
Source: Based on Ashton and Leppard (2005).
 
Owing to the wide-ranging nature of lesions, it is helpful to understand their different types. Therefore, specific definitions and clinical examples of particular surface features and lesions are now summarised in Table 3.4.


   
 
Table 3.4 Types and definitions: Surface features and lesions.

 Types of lesion 
 NormalSmooth, the absence of other surface features
 ScalyExcess dead epidermal scales produced by shedding from the stratum corneum or abnormal keratinisation (e.g. erythrodermic psoriasis)
 HyperkeratoticIncreased keratinisation (cornification) of the epidermis, which appears clinically as thickened and rough skin or mucous membrane (e.g. foot psoriasis)
 WartyA wart-like lesion consisting of finger-like projections (e.g. filiform wart)
 CrustDried exudate (comprised of dried serum, bacteria and possibly blood, mixed with epidermal debris – e.g. impetigo)
 ExcoriatedA superficial linear erosion caused by excessive scratching (e.g. atopic eczema)
 ExudateA leakage of fluid from blood vessels into nearby tissue (e.g. acute eczema)
 Flat: maculeA flat lesion circumscribed area of altered skin colour <1 cm in diameter (e.g. vitiligo, solar lentigo)
 Flat: patchA flat lesion >1 cm in diameter (e.g. port wine stain)
 Raised: papuleA raised lesion <1 cm in diameter (e.g. compound naevus)
 Raised: plaqueA slightly raised flat-topped lesion >1 cm in diameter of surface skin (e.g. plaque psoriasis, pityriasis rosea)
 Raised: noduleA solid palpable mass that is larger than 1 cm whose greater part lies beneath the skin (e.g. erythema nodosum, basal cell carcinoma)
 Fluid-filled: vesicleA small lesion <5 mm in diameter, fluid-containing elevation (e.g. herpes simplex, eczema herpeticum)
 Fluid-filled: bullaeA lesion >5 mm in diameter, fluid-containing elevation (e.g. bullous pemphigoid)
 Fluid-filled: pustuleA lesion <1 cm filled with pus (e.g. acne vulgaris)
 Due to broken surface: ulcerLoss of epidermis and dermis (e.g. ducibitus [pressure] ulcer)
 Due to broken surface: erosionLoss of epidermis only (e.g. intertrigo - a rash in body folds)
 Due to broken surface: fissureLinear split in skin: foot psoriasis (e.g. a heel fissure)
 Colour: due to bloodPetechia (pin head size) (e.g. Meningococcal disease – that do not disappear when pressure if applied) – they are purpuric lesions up to 2 mm across; Purpura (<2.5 mm): red, purple or orange/brown colour due to blood leaking from blood vessels (does not blanche under pressure) (e.g. drug eruption, allergic vasculitis); Haematoma (bruise); Telangiectasia: spider-like capillaries (e.g. due to chronic treatment with topical corticosteroids)
 Colour: due to pigmentMay be due to increase in melanin pigment following epidermal inflammation (e.g. lichen planus)
 Colour: due to lack of blood/pigmentDepigmentation: complete loss of melanin (e.g. vitiligo) Hypopigmentation: partial melanin loss due to epidermal inflammation (e.g. eczema)
 Colour: otherYellow (e.g. xanthelasma)
 
   

A useful distinction is also made of primary and secondary lesions. Primary lesions are caused directly by the disease process; this includes macules, papules, nodules, plaques, wheals, vesicles, bulla, pustules and cysts (Figures 3.3–3.8). Secondary lesions refer to the consequences of the disease process; these include scale, crust, fissures, lichenification, erosion, ulcers, excoriation, scar and atrophy (Johannsen, 1998). Further details of physical signs in dermatology, with excellent illustrative photographs, can be found in Lawrence and Cox (2002). Many skin diseases have a classic distribution which aids diagnosis; this is illustrated in Figure 3.9.


Figure 3.3 Macule. (Source: Reprinted from Graham - Brown and Burns, 2006.)
Figure 3.3 Macule. (Source: Reprinted from Graham - Brown and Burns, 2006.)
 
Figure 3.4 Plaque. (Source: Reprinted from Graham - Brown and Burns, 2006.)
Figure 3.4 Plaque. (Source: Reprinted from Graham - Brown and Burns, 2006.)

Figure 3.5 Papule. (Source: Reprinted from Graham - Brown and Burns, 2006.)
Figure 3.5 Papule. (Source: Reprinted from Graham - Brown and Burns, 2006.)
 
Figure 3.6 Vesicle. (Source: Reprinted from Graham - Brown and Burns, 2006.)
Figure 3.6 Vesicle. (Source: Reprinted from Graham - Brown and Burns, 2006.)

Figure 3.7 Bullae. (Source: Reprinted from Graham-Brown and Burns, 2006.)
Figure 3.7 Bullae. (Source: Reprinted from Graham-Brown and Burns, 2006.)
 
Figure 3.8 Nodule. (Source: Reprinted from Graham-Brown and Burns, 2006.)
Figure 3.8 Nodule. (Source: Reprinted from Graham-Brown and Burns, 2006.)

Figure 3.9 Classic lesion distribution in common skin disorders (Mackie, 2003).
Figure 3.9 Classic lesion distribution in common skin disorders (Mackie, 2003).